Endothelial cells are prone to inflammation under many other conditions.
Smoking. Autoimmune disease. Insulin resistance. And, of course, high levels of apoB.
And while transcytosis of the apoB particle can occur with a perfectly healthy endothelium, the inner lining of the arterial walls, some apoB particles effortlessly glide through the endothelium and transform into a ticking time bomb that threatens our cardiovascular health. How so?
Dr Thomas Dayspring has a thorough explanation beginning with the macrophages (white blood cells) that become scavengers capable of engulfing substances.
Tune in to learn more about how endothelial dysfunction causes atherosclerosis and why high levels of apoB are always NOT a good sign despite the absence of inflammation.
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