DILATED CARDIOMYOPATHY PATHOPHYSIOLOGY
Dilated cardiomyopathy, and pretty much every other cardiovascular disorder you'll learn about in nursing school, can be tricky to understand.
In this video, I'll give you a step-by-step breakdown of the pathophysiology of dilated cardiomyopathy.
Dilated cardiomyopathy is one of the types of cardiomyopathy. During dilated cardiomyopathy, the heart is weakened and it can't pump blood as easily. This reduces the cardiac output of the heart, meaning there is less blood being pumped out to the body.
During this type of cardiomyopathy, the walls of the heart dilate (or thin), which further decreases the heart's ability to pump blood out. So the heart just gets weaker and weaker.
Here the step-by-step breakdown of dilated cardiomyopathy pathophysiology:
1. There is a trigger that weakens the heart. There is something going on in the body that weakens the heart and causes the walls of the heart to dilate.
2. Decreased cardiac output. There is less blood going to the body.
3. The heart walls dilate more to try to get more and more blood out to the body. The heart is thinking, "If I could just expand my walls a little bit more, maybe I can fit more blood inside my ventricles, and push more blood out."
This constant dilating of the walls of the heart weakens it more and more, causing the cardiac output to decrease more.
And that is the pathophysiology of dilated cardiomyopathy!
If this explanation of dilated cardiomyopathy makes sense to you, write "LOVE" in the comments below.
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